Expanding the continuum of substance use disorder treatment: Nonabstinence approaches PMC

milestone 04-02-2020
Expanding the continuum of substance use disorder treatment: Nonabstinence approaches PMC

For example, the CBT intervention developed in Project MATCH [18] (described below) equated to RP with respect to the core sessions, but it also included elective sessions that are not typically a focus in RP (e.g., job-seeking skills, family involvement). Cognitive behaviour therapy is a structured, time limited, psychological intervention that has is empirically supported across a wide variety of psychological disorders. CBT for addictive behaviours can be traced back to the application of learning theories in understanding addiction and subsequently to social cognitive theories. The focus of CBT is manifold and the focus is on targeting maintaining factors of addictive behaviours and preventing relapse.

Despite this, lapsing is still a risk factor and makes a person more prone to relapse. What is more, negative feelings can create a negative mindset that erodes resolve and motivation for change and casts the challenge of recovery as overwhelming, inducing hopelessness. A relapse or even a lapse might be interpreted as proof that a person doesn’t have what it takes to leave addiction behind. Cravings can be dealt with in a great variety of ways, and each person needs as array of coping strategies to discover which ones work best and under what circumstances. Another is to carefully plan days so that they are filled with healthy, absorbing activities that give little time for rumination to run wild.

Does 12-Step Contribute to the AVE?

A good treatment program should explain the difference between a lapse and relapse. It should also teach a person how to stop the progression from a lapse into relapse. Marlatt’s relapse prevention model also identifies certain factors called covert antecedents which don’t stand out as clearly. Examples include denial, rationalization of why it’s okay to use (i.e. to reduce stress), and/or urges and cravings. Clinicians in relapse prevention Alcoholic ketoacidosis Wikipedia programs and the field of clinical psychology as a whole point out that relapse occurs only after a long-term pattern of specific feelings, thoughts, and behavior. A more recent development in the area of managing addictive behaviours is the application of the construct of mindfulness to managing experiences related to craving, negative affect and other emotional states that are believed to impact the process of relapse34.

The study was especially notable because most other treatment readiness measures have been validated on treatment-seeking samples (see Freyer et al., 2004). This finding supplements the numerous studies that identify lack of readiness for abstinence as the top reason for non-engagement in SUD treatment, even among those who recognize a need for treatment (e.g., Chen, Strain, Crum, & Mojtabai, 2013; SAMHSA, 2019a). The harm reduction movement, and the wider shift toward addressing public health impacts of drug use, had both specific and diffuse effects on SUD treatment research. In 1990, Marlatt was introduced to the philosophy of harm reduction during a trip to the Netherlands (Marlatt, 1998). He adopted the language and framework of harm reduction in his own research, and in 1998 published a seminal book on harm reduction strategies for a range of substances and behaviors (Marlatt, 1998).

Combatting the Abstinence Violation Effect

Working with a variety of targets helps in generalization of gains, patients are helped in anticipating high risk situations33. The individual’s reactions to the lapse and their attributions (of a failure) regarding the cause of lapse determine the escalation of a lapse into a relapse. The https://trading-market.org/dedicated-to-life-long-recovery/ is characterized by two key cognitive affective elements.

The model incorporates the stages of change proposed by Procahska, DiClement and Norcross (1992) and treatment principles are based on social-cognitive theories11,29,30. Cognitive behaviour therapy (CBT) is a structured, time limited, evidence based psychological therapy for a wide range of emotional and behavioural disorders, including addictive behaviours1,2. CBT belongs to a family of interventions that are focused on the identification and modification of dysfunctional cognitions in order to modify negative emotions and behaviours.

Models of nonabstinence psychosocial treatment for SUD

Marlatt, in particular, became well known for developing nonabstinence treatments, such as BASICS for college drinking (Marlatt et al., 1998) and Relapse Prevention (Marlatt & Gordon, 1985). Like the Sobells, Marlatt showed that reductions in drinking and harm were achievable in nonabstinence treatments (Marlatt & Witkiewitz, 2002). In the 1970s, the pioneering work of a small number of alcohol researchers began to challenge the existing abstinence-based paradigm in AUD treatment research. They found that their controlled drinking intervention produced significantly better outcomes compared to usual treatment, and that about a quarter of the individuals in this condition maintained controlled drinking for one year post treatment (Sobell & Sobell, 1973).

abstinence violation effect

A careful functional analysis and identification of dysfunctional beliefs are important first steps in CBT. The hallmark of CBT is collaborative empiricism and describes the nature of therapeutic relationship. There are no specific time frames within which a person navigates through the stages, and may also remain at stage for a long time before moving forwards or backwards (for example a person may remain in the stage of contemplation or preparation for years without moving on to action). Patterns of movement through the various stages are categorized as stable, progressive or unstable11. It is hoped that more severely mentally ill people will obtain life-saving treatment and pathways to better housing. If you are at a gathering where provocation arises because alcohol or other substances are available, leave.

2. Established treatment models compatible with nonabstinence goals

Efforts to develop, test and refine theoretical models are critical to enhancing the understanding and prevention of relapse [1,2,14]. A major development in this respect was the reformulation of Marlatt’s cognitive-behavioral relapse model to place greater emphasis on dynamic relapse processes [8]. Whereas most theories presume linear relationships among constructs, the reformulated model (Figure ​(Figure2)2) views relapse as a complex, nonlinear process in which various factors act jointly and interactively to affect relapse timing and severity. Similar to the original RP model, the dynamic model centers on the high-risk situation. Against this backdrop, both tonic (stable) and phasic (transient) influences interact to determine relapse likelihood. Tonic processes include distal risks–stable background factors that determine an individual’s “set point” or initial threshold for relapse [8,31].

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